Heart Attack (Myocardial Infarction)

What is Heart Attack (Myocardial Infarction)?

The heart muscle gets its own blood through small arteries on its surface, the coronary arteries. Over years, fatty plaque can build up in their walls.

If a plaque cracks, a blood clot forms on it and can block the artery. The muscle beyond it gets no oxygen and begins to die. Doctors call this a myocardial infarction — heart muscle that has died because its blood stopped. The sooner the artery is opened, the more muscle is saved.

What are the symptoms?

• Chest pain, pressure or squeezing, usually lasting more than a few minutes
• Pain spreading to the arm, jaw, neck, shoulder or back
• Shortness of breath
• Cold sweat, nausea, vomiting, or a feeling like indigestion
• Dizziness, light-headedness or fainting
• Unusual tiredness — more common in women; some heart attacks are silent

What causes it, and who is more likely to get it?

How is it found?

A heart attack is diagnosed in hospital, fast — the tests run while treatment begins.

A neurotherapist never treats chest pain on the mat. After the hospital has treated the heart attack, the therapist asks for the discharge summary, the echo report with its ejection fraction, and every medicine — especially blood thinners — and checks whether any wound is still open.

How Neurotherapy sees Heart Attack (Myocardial Infarction)

LMNT reads an infarct as a problem of clots. Its teaching gives two words to keep apart: ischaemia is hunger — the part is alive but underfed; an infarct is a death in the tissue — the part died because the blood stopped. Blood is safe while it moves; slow it or stop it inside a vessel and it begins to set. The body has its own answer: it makes heparin to stop unwanted clots, and plasmin to dissolve clots already formed.

How the body opens a clot

1. Clotalready holds a waiting chemical, plasminogen
2. Vessel lininghours after an injury, releases tPA
3. PlasmintPA slowly turns plasminogen into plasmin — in about 24 hours
4. Fibrinplasmin digests the fibrin threads that hold the clot together
5. Flowthe clot opens; if this fails, LMNT teaches, clots keep growing

P. Heparin — (8) Pan (7) Liv (8) Ch. Only — was built in 1994 with two aims: that unwanted clots should not form, and that old clots should be dissolved. LMNT says it can be given to all patients and lists the heart among its targets. The therapy reports the ejection fraction rising over several months of P. Heparin — a clinical observation measured on the cardiologist's scan, not a trial result.

Neurotherapy treatment for Heart Attack (Myocardial Infarction), step by step

1. Emergency first

   Chest pain or pressure: call 108/112 and do not drive yourself. LMNT is never first aid for a heart attack; it comes only in recovery, after the hospital's treatment.

2. Examine first

   The therapist checks Liv° and Mu°, reads the discharge summary and echo report, notes every medicine, and asks whether any wound — such as an angiography or surgery site — is still open.

   Liv°Mu°

3. P. Heparin

   The pancreas point first, to help digest the fibrin of old clots; then the liver and chest points, to help the body make its own heparin. Clear first, then prevent.

   P. Heparin (plasmin)

4. Let the reports speak

   Progress is judged by the cardiologist's repeat tests, such as the echo, not by a hand. The heart team decides every change in medicines.

Points and formulas used

Watch

P HAPARIN FORMULA

Shows the P. Heparin formula: (8) Pan at the top of the thighs, (7) Liv on the thigh and forearm, and (8) Chest Only with the palms on the chest. In the video it is given as the last part of the Angina treatment, to support circulation.

The P. Heparin formula — for recovery after hospital treatment, never as first aid for chest pain.

From Ayurveda and the kitchen

Test yourself

For you

Related topics